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dc.contributor.authorSuryavanshi, Santosh
dc.contributor.authorAnderson, Kody
dc.contributor.authorJadhav, Shweta
dc.contributor.authorLichtarge, Olivier
dc.contributor.authorMcConnell, Bradley K.
dc.date.accessioned2018-02-23T19:36:23Z
dc.date.available2018-02-23T19:36:23Z
dc.date.issued2017
dc.identifier.urihttp://hdl.handle.net/10657/2340
dc.description.abstractmAKAP polymorphisms may pre-dispose humans to CVDs by altering cAMP/PKA signaling in the heart. PKA-mAKAP interaction modulators can be developed as therapeutic target to augment current treatment of CVDs. This project was completed with contributions from Alexei Degterev from the Department of Developmental, Molecular and Chemical Biology, School of Medicine, Tufts University, Boston.
dc.language.isoen_US
dc.titleHuman Muscle-Specific A-Kinase Anchoring Protein (mAKAP) Polymorphisms: Mechanistic Role in Cardiovascular Diseases
dc.typePoster
dc.description.departmentPharmacological and Pharmaceutical Sciences, Department of


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  • GRS 2017
    This collection gathers posters, talks, and performances presented at the 2017 Graduate Research and Scholarship Projects Day

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